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HFCS Influence on Insulin Resistance Discovered

Study:  Specific Gene Produces Excess Fat with HFCS Consumption

Soda contains high fructose corn syrup

Soda contains high fructose corn syrup

A talk radio program addressed something the other day that I’ve seldom heard anyone outside of health professionals on television or in print mention:  the ubiquity of high fructose corn syrup in every day foods.

Certainly you’ve noticed this.  Read any food label – even foods that are considered to be healthy, like l bran flakes or wheat flakes – and one of the top ingredients is the stuff most have shortened to four letters (HFCS). 

Is it really any wonder, then, why obesity levels are as high as they are?  Or why diabetes diagnoses have jumped an astounding 90 percent in the past 10 years? 

As I’ve documented here and as has been documented elsewhere, high fructose corn syrup is one of the more toxic sweeteners known to man because of the way in which the body metabolizes it.  It’s converted to fat very, very quickly, and when consumed regularly over the course of days, weeks, months and years, it takes a significant toll on the body, leading to obesity and insulin resistance, a precursor to type II diabetes and all that it entails.

The question, of course, is why high fructose corn syrup is so quick to turn to fat than, say, glucose?  Well, researchers believe they may have found the answer to that question after putting a group of insulin-resistant rats through a series of tests. 

As reported in the medical journal Cell Metabolism this month, more than two dozen researchers from some of the country’s finest medical institutions and universities took part in a study that honed in on how HFCS affected the chemical processes of insulin-resistant rats.  They came away with many findings, but the main one was that HFCS appear to influence a specific gene – called PPARg co-activator 1b, if you’re curious. This gene produces an excessive amount of fat that the liver stores when HFCS are consumed.  But when researchers manipulated the gene so that it wouldn’t be affected by HFCS consumption – essentially blocking the gene from producing fat – fat stores declined and the rats’ insulin resistance subsided significantly.

Certainly this is a breakthrough find, but all it really does is satiate chemically-minded brains on why certain things affect the body the way they do.  In other words, it doesn’t get us any closer to becoming resistant to insulin resistance.

Insulin resistance may indeed be an inherited trait, but in an increasing number of cases (I would argue in the majority of cases, but I have no numbers to back that up), it’s inherited due to one’s diet. 

Because of HFCS prevalence, you really need to be careful of what you’re eating at all times and scan the labels whenever you’re not reaching for an organic product.  Because the organic seal is the only thing that assures what you’re buying doesn’t have a trace of high fructose corn syrup.

Each American consumed an estimated 60 pounds worth of high fructose corn syrup in 2005!   The only way to decrease that total for 2009 and beyond is by scanning the labels for each and every thing you buy.  If that sounds like a pain – and it sure does to me – the time is now to go organic.    

Sources:
Chicago Tribune
ScienceDaily

Related Posts

  1. High Fructose Equals No Glucose? Study Says High Fructose Corn Syrup Can Lead to Diabetes
  2. 7UP Soda No Longer to be Marketed as “All-Natural”…
  3. “Gene” Made Me Do It: Could ‘FTO’ Gene Be Reason Why We Overeat?
  4. Pregnant Women Taking Nutritional Supplements Positively Influence Future Generations
  5. Artificial Food Additives Increase Hyperactivity Risk in Children, Study Finds
  


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